Publication2017
Nanoquinacrine caused apoptosis in oral cancer stem cells by disrupting the interaction between GLI1 and β catenin through activation of GSK3β
The record
- Type
- Publication
- Date
- 1 September 2017
- Level
- National
- Journal
- Toxicology and Applied Pharmacology
- Volume
- 330
- Pages
- 53-64
- Published
- 1 September 2017
- DOI
- 10.1016/j.taap.2017.07.008
- Scopus ID
- 85024901072
- ISSN
- 0041008X
- Authors
- 6
- Open access
- No
- Citations
- 20
- International co-authorship
- No
- Co-author countries
- IN
Classification
Every label below carries the reasoning it was assigned on, the confidence band it was assigned in, and what produced it. The rubric is published.
UN Sustainable Development Goals
3.3 End the epidemics of AIDS, tuberculosis, malaria, neglected tropical diseases, hepatitis and water-borne diseases
Nanoquinacrine-induced apoptosis in oral cancer stem cells through pathway disruption addresses cancer cell death mechanisms and therapeutic strategies.
- High confidence
- The subject matter is a strong and direct inference from the record type, the funding or organising body, or the programme context, even where the title alone is thin. Example: an ICMR-funded project against SDG 3, on the strength of the agency.
- Judged
- Judged against the published classification rubric, under the rubric and prompt versions recorded with the run that produced it.
- Produced by
- claude-haiku-4-5-20251001, under kortex-publication-v1
Viksit Bharat @2047
vb-1.2 Health and well-being
Oral cancer therapy research directly improves health and well-being through novel drug mechanisms.
- Produced by
- claude-haiku-4-5-20251001, under kortex-publication-v1
Each label above carries the instrument and the version that produced it. How each classification is reached.
Evidence
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Provenance
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- Source rows resolved into this record
- 1
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Citing this record
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KIIT Deemed to be University. “Nanoquinacrine caused apoptosis in oral cancer stem cells by disrupting the interaction between GLI1 and β catenin through activation of GSK3β”. Kortex.