Publication2017
Increasing NO level regulates apoptosis and inflammation in macrophages after 2-chloroethyl ethyl sulphide challenge
- Good Health and Well-being
- Empowered Indians
The record
- Type
- Publication
- Date
- 1 February 2017
- Level
- National
- Journal
- International Journal of Biochemistry and Cell Biology
- Volume
- 83
- Pages
- 1-14
- Published
- 1 February 2017
- DOI
- 10.1016/j.biocel.2016.12.004
- Scopus ID
- 85002741716
- ISSN
- 13572725
- Authors
- 7
- Open access
- No
- Citations
- 20
- International co-authorship
- No
- Co-author countries
- IN
Classification
Every label below carries the reasoning it was assigned on, the confidence band it was assigned in, and what produced it. The rubric is published.
UN Sustainable Development Goals
3 Ensure healthy lives and promote well-being for all at all ages
Demonstrates nitric oxide mechanisms regulating apoptosis and inflammatory responses in macrophages following chemical exposure stress.
- Moderate confidence
- The alignment is plausible and defensible but rests on an assumption a reasonable reviewer could decline. Published, and surfaced in the review queue for a human to confirm or override.
- Judged
- Judged against the published classification rubric, under the rubric and prompt versions recorded with the run that produced it.
- Produced by
- claude-haiku-4-5-20251001, under kortex-publication-v1
Viksit Bharat @2047
vb-1 Empowered Indians
Derived through the seeded SDG-to-Viksit-Bharat crosswalk from the primary SDG 3 label. Demonstrates nitric oxide mechanisms regulating apoptosis and inflammatory responses in macrophages following chemical exposure stress.
- Produced by
- claude-haiku-4-5-20251001, under kortex-publication-v1
Each label above carries the instrument and the version that produced it. How each classification is reached.
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- Source rows resolved into this record
- 1
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Citing this record
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KIIT Deemed to be University. “Increasing NO level regulates apoptosis and inflammation in macrophages after 2-chloroethyl ethyl sulphide challenge”. Kortex.